Wednesday, April 7, 2021

(Revised 7/6/2021)

(Advisory: See Update to Article, The Zika Virus Scare: An Anatomy Of How Diseases Are Manufactured For Political Objectives)

The Marxist Establishment Lies By Omission: mRNA Vaccines And Your Proprietary Genetic Code

                       Missing from illustration: An RNA polymerase (in the nucleus) transcribes DNA into                                                                       a messenger RNA

Pfizer, Moderna, and AstraZeneca 'COVID-19' 'vaccines' aren't vaccines. A vaccine uses a weakened strain of the pathogen. In the case of Pfizer, Moderna, and AstraZeneca serums, an mRNA enzyme is used sending foreign instructions for a new protein to be added to one's prospective DNA code:

How Vaccines Work

When a person is infected with a germ, whether a virus or bacteria, the immune system creates special proteins, called antibodies, that help protect against future infections from that germ. The next time your immune system sees that germ, it "remembers" and uses the antibodies to fight against the infection. Some antibodies only last a few months, while others can protect you for a lifetime.

Vaccines create antibodies that allow your body to protect itself from future infections without actually getting sick.

Previously developed vaccines contain very small amounts of viruses or bacteria that are dead or greatly weakened. They trick the immune system into believing that the body is being infected.

The COVID vaccine is no different in that it creates antibodies, but it uses a different set of tricks than traditional vaccines to create coronavirus immunity.

How mRNA Vaccines Work

mRNA is a piece of genetic material that cells use as "instructions" to create certain proteins in the body. It is like a bit of computer code.

When it's not inside a cell, mRNA needs protection to keep it from disintegrating. This is why the vaccines require cold temperature storage. To keep the mRNA from disintegrating when it enters the body, the COVID-19 vaccines use fat bubbles to shuttle the mRNA to certain cells.

The mRNA instructs these cells to create "spike proteins." These proteins simulate part of the SARS-CoV-2 (novel coronavirus) cell structure and trick the body into believing it's infected with the virus.

In the case of the mRNA vaccines, your body is never exposed to the germ but is still able to produce an effective immune response.

The article fails to mention any concerns for DNA alteration by mRNA 'vaccines', so let's take a look at what the CDC has to say about mRNA ‘vaccines’:

Facts about COVID-19 mRNA Vaccines

They cannot give someone COVID-19.

• mRNA vaccines do not use the live virus that causes COVID-19.

They do not affect or interact with our DNA in any way.

• mRNA never enters the nucleus of the cell, which is where our DNA (genetic material) is kept.
• The cell breaks down and gets rid of the mRNA soon after it is finished using the instructions.

Is the second bullet point true? Yes it is, the mRNA enzyme administered doesn’t enter the nucleus, it can only enter a ribosome that is located in the cytoplasm outside of the nucleus, in order to provide the instructions to the ribosome for the manufacture of the weakened “spike protein” that’s supposed to activate the body’s immune response. mRNA enzymes only leave the nucleus (heading for the ribosomes), so when a foreign mRNA is introduced via inoculation, it too is headed for the ribosomes.

So we have nothing to worry about, then, right? No. You see, the CDC committed the sin of omission in telling us that the cell’s genetic code won’t be affected by mRNa. Well, that cell’s genetic code won’t be affected by mRNA, but when a cell divides the genetic code of both the daughter cells will be affected by mutated proteins created in the cytoplasm, where “spike protein” fragments* join with other degraded proteins that will mutate the prospective genetic codes of those daughter cells after [ribosome] protein synthesis/manufacture takes place. 

Mutations That Occur Immediately After Protein Synthesis In The Cytoplasm: Mutated Proteins Will Assist In DNA Replication During Cell Division 

Errors in posttranslational modification are likely important but their frequency and effects remain largely unknown.**

With cells now being flooded with posttranslational "spike protein" fragments, it is indeed certain that errors are taking place, where "spike protein" fragments are linking up with at most a few percent of newly synthesized proteins [that] were rapidly degraded.*** Some of these novel hybrid proteins will be errantly used for the genetic coding of daughter cells; viral integration into human DNA is nothing new, but hybrid proteins forming from viral and human protein fragments is mutagenic.  

In the following video, Dr. Mobeen Syed not only provides a comprehensive overview on how precisely mRNA 'vaccines' work, the mechanics, he also illustrates the various antigen proteins that destroy loose DNA, RNA and proteins within tissue. Dr. Syed also informs us that once a cell is seen by the immune system to be infected with foreign fragments, the immune system will destroy the host cell. In this case the foreign objects are spike protein fragments that haven't yet been transferred out of the host cell to the cell's outer envelop where T cells work on the fragments to create the future immune response. All good and well, and very informative. But this is how cells react in a perfect environment where the pathogen introduction is limited in scope. What will happen when a localized group of cells are massively overloaded, as is the case when the upper arm is jabbed with a massive infusion of foreign pathogens? Then two weeks later the same infusion quantity? Many spike protein fragment carrying cells are going to escape destruction, allowing for subsequent replication. So what, you say? Well, the problem is with denatured (incomplete) proteins commonly cropping up in cell's cytoplasm after ribosome manufacture. Such denatured proteins can hook up with denatured spike protein fragments, becoming complete 'hybrid proteins' involved in the replication of DNA. Not DNA itself, but protein enzymes that guide the replication process. For example, when a DNA strand separates into two in order to create two new cells, it's a protein enzyme that begins the process. Other proteins assist in assembling the new DNA  strands. These hybrid proteins then will result in miscoding of one's DNA during cell reproduction.

The CDC informs that...

mRNA never enters the nucleus of the cell, which is where our DNA (genetic material) is kept.

That's true, but isn't the threat. The threat comes after the mRNA template is translated by ribosomes into spike proteins, then when the spike proteins are chopped up into denatured fragments by a cell's proteasomes, those spike protein fragments can hook up with denatured proteins that were slated for DNA replication.

Back to the CDC article. It reads:

mRNA vaccines have been studied before for flu, Zika, rabies, and cytomegalovirus (CMV).

We’ll stop there, because we needn’t go any further when Zika is brought up as a reassuring testimonial for mRNA “vaccines”. Unlike COVID-19 that doesn't exist, the Zika virus exists but doesn't cause microcephaly, which is easily determined thanks to knowing how to count. Yes, it’s that simple, whereas the non-existence of COVID-19 is more varied and therefore more complex to determine.

My article on the Zika Virus-microcephaly scandal (The Zika Virus Scare: An Anatomy Of How Diseases Are Manufactured For Political Objectives) was the first and only in-depth account of how numbers were blatantly and obviously confounded:

In 2014, under a non-reporting requirement regime, Brazilian physicians reported a fraction of microcephaly cases for that year; 147 cases to be exact. Under the new reporting regime that took over for 2015, the number of cases naturally increased massively, the numbers representing what would be expected under a reporting regime. Then using the artificially low 2014 numbers as the baseline, Brazil and the global political/medical community (though not all within the medical community) declared an epidemic of microcephaly in Brazil based on the higher, though expected, numbers under the new reporting requirement regime. This obvious charade brought about a few well-known epidemiologists and physicians to sound the alarm, such as Alexandre Dias Porto Chiavegatto Filho, professor of epidemiology at the University of Sao Paulo: 

It's a global scandal. Brazil has created a worldwide panic. 

Firstly, it's the global community, not just Brazil, that's playing fast and loose with the fake Zika virus epidemic, and secondly, there never was a reason to believe that Brazil was suffering an epidemic of microcephaly in the first place, since it was well known that pre-2015 Brazilian doctors only reported a small fraction of such cases to the central government; pre-2015 there existed no reporting requirement for microcephaly in Brazil. The sudden emergence in 2015 of an 'epidemic' of microcephaly in Brazil actually represents numbers that would be expected given a comprehensive reporting requirement.

Addendum

DNA alteration by the spike protein causes long-term diseases, but sort-term diseases also occur: 

Within a cell, after ribosomes turn the mRNA spike protein into a spike protein, the cell's proteasomes tears the spike protein into fragments. Some of these fragments are carried out of the cell by mhc1 to attach to the cell membrane. Later, when the infected cell is destroyed, spike protein fragments are let loose and attach to other cells' membranes. Here's the problem: When the spike protein is attached to an actual virus, it allows for entry into a cell. In the case of spike protein fragments that are let loose after a cell is destroyed, the fragment (1) doesn't want to enter the cell; allowing the fragment (2) to link up with another cell that collides with the fragment; thereby (3) creating multicell groupings; that (4) produce blood clots and their related pathologies.

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* Will Vaccine Generated Spike Proteins Bind To Our Cells?

(fast-forward in video to 12:15 minutes) 

Protein Synthesis and Mutations

In the video, "Will Vaccine Generated Spike Proteins Bind To Our Cells?", Dr. Mobeen Syed assumes T cell destruction of infected cells to be 100%. This is not the case:

SUMMARY

According to in vitro assays, T cells are thought to kill rapidly and efficiently, but the efficacy and dynamics of cytotoxic T lymphocyte (CTL)-mediated killing of virus-infected cells in vivo remains elusive. Weused two-photon microscopy to quantify CTL-mediated killing in mice infected with herpesviruses or poxviruses. On average, one CTL killed 2–16 virusinfected cells per day as determined by real-time imaging and by mathematical modeling. In contrast, upon virus-induced MHC class I downmodulation, CTLs failed to destroy their targets. During killing, CTLs remained migratory and formed motile kinapses rather than static synapses with targets. Viruses encoding the calcium sensor GCaMP6s revealed strong heterogeneity in individual CTL functional capacity. Furthermore, the probability of death of infected cells increased for those contacted by more than two CTLs, indicative of CTL cooperation. Thus, direct visualization of CTLs during killing of virus-infected cells reveals crucial parameters of CD8+ T cell immunity.

Infected cells that survive T cell attack, the "spike protein" fragments within the cell can then join with another protein, thereby producing a hybrid protein - a mutation. Wherever those hybrid proteins end up, some will end up back in the nucleus to be included in the DNA structures of prospective daughter cells once the parent cell has divided.

** TRANSCRIPTION, TRANSLATION AND REPLICATION

***  During protein synthesis in a ribosome, START and STOP codons are only required for mRNA. With protein fragments, however, there may be no codon for START or STOP; beginning or stopping protein synthesis. There is one such codon that’s read by tRNA for START: AUG. Three codons are read by tRNA as STOP: UAG, UAA, and UGA. 

When one such codon on the mRNA stand is read by the translation molecule (tRNA), the manufacture of that protein either commences (START) or comes to an end (STOP). Therefore when fragments from different proteins collide after leaving their respective ribosome, they can join becoming one mutagenic protein: e.g., a protein fragment with a START codon but no STOP codon hooks up with a fragment from a different protein that has no START codon but does have a STOP codon:

Small protein fragments, and not just residues, can be used as basic building blocks to reconstruct networks of coevolved amino acids in proteins. Fragments often enter in physical contact one with the other and play a major biological role in the protein. The nature of these interactions might be multiple and spans beyond binding specificity, allosteric regulation and folding constraints. Indeed, coevolving fragments are indicators of important information explaining folding intermediates, peptide assembly, key mutations with known roles in genetic diseases [emphasis mine], distinguished subfamily-dependent motifs and differentiated evolutionary pressures on protein regions.